Refining the evidence for GSTA1 and eNOS genetic effects on risk of radiotherapy-induced telangiectasia

Int J Cancer. 2008 Dec 15;123(12):2973-4. doi: 10.1002/ijc.23854.
No abstract available

Publication types

  • Comment
  • Letter

MeSH terms

  • Breast Neoplasms / radiotherapy*
  • Female
  • Glutamic Acid
  • Glutathione Transferase / genetics*
  • Humans
  • Logistic Models
  • Nitric Oxide Synthase Type III / genetics*
  • Polymorphism, Single Nucleotide*
  • Radiation Injuries / enzymology
  • Radiation Injuries / etiology*
  • Radiation Injuries / genetics
  • Radiotherapy / adverse effects
  • Risk Assessment
  • Risk Factors
  • Telangiectasis / enzymology
  • Telangiectasis / etiology*
  • Telangiectasis / genetics
  • Vasodilation / genetics

Substances

  • Glutamic Acid
  • NOS3 protein, human
  • Nitric Oxide Synthase Type III
  • GSTA1 protein, human
  • Glutathione Transferase