Treatment with LXR agonists after focal cerebral ischemia prevents brain damage

FEBS Lett. 2008 Oct 15;582(23-24):3396-400. doi: 10.1016/j.febslet.2008.08.035. Epub 2008 Sep 19.

Abstract

Stroke is characterized by massive inflammation in areas surrounding the injury that magnifies damage to the brain. The liver X receptors (LXRs) are nuclear receptors that regulate cholesterol, lipid, and glucose metabolism. Synthetic LXR agonists have potent anti-inflammatory properties in a variety of settings, including neuroinflammation. However, the ability of LXR agonists to suppress stroke-associated inflammation has not been evaluated. Here, we have used time-lapse magnetic resonance imaging (MRI) to show that a single dose of an LXR ligand administered post-injury dramatically reduces brain damage in a model of acute brain ischemia. Neuroprotection was associated with suppression of neuroinflammation.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Animals
  • Benzoates / therapeutic use*
  • Benzylamines / therapeutic use*
  • Brain Ischemia / complications
  • Brain Ischemia / drug therapy*
  • Brain Ischemia / pathology
  • Cerebral Infarction / etiology
  • Cerebral Infarction / pathology
  • Cerebral Infarction / prevention & control*
  • DNA-Binding Proteins / agonists*
  • Disease Models, Animal
  • Ligands
  • Liver X Receptors
  • Magnetic Resonance Imaging
  • Male
  • Orphan Nuclear Receptors
  • Rats
  • Rats, Sprague-Dawley
  • Receptors, Cytoplasmic and Nuclear / agonists*

Substances

  • Benzoates
  • Benzylamines
  • DNA-Binding Proteins
  • GW 3965
  • Ligands
  • Liver X Receptors
  • Orphan Nuclear Receptors
  • Receptors, Cytoplasmic and Nuclear