FMRFamide neuropeptides and acetylcholine synergistically inhibit egg-laying by C. elegans

Nat Neurosci. 2008 Oct;11(10):1168-76. doi: 10.1038/nn.2186. Epub 2008 Sep 21.

Abstract

Egg-laying behavior of the Caenorhabditis elegans hermaphrodite is regulated by G protein signaling pathways. Here we show that the egg laying-defective mutant egl-6(n592) carries an activating mutation in a G protein-coupled receptor that inhibits C. elegans egg-laying motor neurons in a G(o)-dependent manner. Ligands for EGL-6 are Phe-Met-Arg-Phe-NH(2) (FMRFamide)-related peptides encoded by the genes flp-10 and flp-17. flp-10 is expressed in both neurons and non-neuronal cells. The major source of flp-17 peptides is a pair of presumptive sensory neurons, the BAG neurons. Genetic analysis of the egl-6 pathway revealed that the EGL-6 neuropeptide signaling pathway functions redundantly with acetylcholine to inhibit egg-laying. The retention of embryos in the uterus of the C. elegans hermaphrodite is therefore under the control of a presumptive sensory system and is inhibited by the convergence of signals from neuropeptides and the small-molecule neurotransmitter acetylcholine.

Publication types

  • Research Support, N.I.H., Extramural
  • Research Support, Non-U.S. Gov't

MeSH terms

  • Acetylcholine / metabolism
  • Acetylcholine / pharmacology*
  • Alanine / genetics
  • Animals
  • Animals, Genetically Modified
  • Behavior, Animal / drug effects*
  • Behavior, Animal / physiology*
  • Caenorhabditis elegans
  • Caenorhabditis elegans Proteins / genetics
  • Caenorhabditis elegans Proteins / physiology
  • FMRFamide / metabolism
  • FMRFamide / pharmacology*
  • Female
  • Gene Expression Regulation / drug effects
  • Gene Expression Regulation / physiology
  • Green Fluorescent Proteins / genetics
  • Green Fluorescent Proteins / metabolism
  • Membrane Potentials / drug effects
  • Membrane Potentials / genetics
  • Membrane Potentials / radiation effects
  • Membrane Transport Modulators / metabolism
  • Membrane Transport Modulators / pharmacology*
  • Motor Neurons / metabolism
  • Motor Neurons / physiology
  • Mutation
  • Neural Inhibition / drug effects
  • Neural Inhibition / physiology
  • Patch-Clamp Techniques / methods
  • Peptides / pharmacology
  • Receptors, G-Protein-Coupled / genetics
  • Receptors, G-Protein-Coupled / metabolism
  • Reproduction* / drug effects
  • Reproduction* / genetics
  • Reproduction* / physiology
  • Signal Transduction / drug effects
  • Signal Transduction / genetics
  • Threonine / genetics

Substances

  • Caenorhabditis elegans Proteins
  • Membrane Transport Modulators
  • Peptides
  • Receptors, G-Protein-Coupled
  • Green Fluorescent Proteins
  • Threonine
  • FMRFamide
  • Acetylcholine
  • Alanine