Differential subcellular localization of insulin receptor substrates depends on C-terminal regions and importin beta

Biochem Biophys Res Commun. 2008 Dec 19;377(3):741-6. doi: 10.1016/j.bbrc.2008.09.106. Epub 2008 Oct 1.

Abstract

Insulin receptor substrates (IRSs) play essential roles in signal transduction of insulin and insulin-like growth factors. Previously, we showed that IRS-3 is localized to the nucleus as well as the cytosol, while IRS-1 and 2 are mainly localized to the cytoplasm. In the present study, we found that importin beta directly interacts with IRS-3 and is able to mediate nuclear transport of IRS-3. Importin beta interacted with the pleckstrin homology domain, the phosphotyrosine binding domain and the C-terminal region of IRS-3; indeed all of these fragments exhibited predominant nuclear localization. By contrast, almost no interaction of importin beta with IRS-1 and -2 was observed, and their C-terminal regions displayed discrete spotty images in the cytosol. In addition, using chimeric proteins between IRS-1 and IRS-3, we revealed that the C-terminal regions are the main determinants of the differing subcellular localizations of IRS-1 and IRS-3.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Animals
  • COS Cells
  • Cell Nucleus / metabolism
  • Chlorocebus aethiops
  • Cytosol / metabolism
  • Humans
  • Mutation
  • Phosphotyrosine / metabolism
  • Protein Structure, Tertiary
  • Rats
  • Receptor, Insulin / genetics
  • Receptor, Insulin / metabolism*
  • beta Karyopherins / genetics
  • beta Karyopherins / metabolism*

Substances

  • beta Karyopherins
  • Phosphotyrosine
  • Receptor, Insulin