Augmentor of liver regeneration ameliorates renal tubular epithelial cell injury after rat liver transplantation

Transplant Proc. 2008 Oct;40(8):2696-9. doi: 10.1016/j.transproceed.2008.08.015.

Abstract

Background: Acute renal insufficiency and dysfunction are common complications after clinical liver transplantation. This study examined whether augmentor of liver regeneration (ALR) played a significant role to ameliorate renal tubular epithelial cell injury after liver transplantation.

Methods: Orthotopic liver transplantation was performed from Sprague-Dawley (SD) to SD rats. Twelve recipients were randomly divided into two groups: ALR group (with recombinated human ALR 100 microg/kg . d intramuscular injection postoperation) versus normal saline-treated group (with the same volume of normal saline injected intramuscularly postoperation). Rats were sacrificed at day 3 posttransplantation. Renal morphological changes in recipients were assessed with light microscopy. The expressions of tumor necrosis factor-alpha (TNF-alpha), proliferating cell nuclear antigen (PCNA) and caspase-3 protein and mRNA in the kidney were evaluated by real-time polymerase chain reaction and immunohistochemical staining.

Results: Morphological changes in renal tubular epithelial cells were not significant in either group at day 3 posttransplantation. The intragraft expression of TNF-alpha and caspase-3 was strikingly promoted in the normal saline-treated group and PCNA attenuated compared to the ALR group.

Conclusion: These data suggested that ALR may play a role to reduce renal damage in liver transplant recipients.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Animals
  • Animals, Newborn
  • Biomarkers
  • Epithelial Cells / pathology*
  • Immunohistochemistry
  • Kidney Tubules / injuries
  • Kidney Tubules / pathology*
  • Liver Regeneration*
  • Liver Transplantation / pathology*
  • Liver Transplantation / physiology*
  • Male
  • RNA, Messenger / genetics
  • Rats
  • Rats, Sprague-Dawley
  • Tumor Necrosis Factor-alpha / genetics

Substances

  • Biomarkers
  • RNA, Messenger
  • Tumor Necrosis Factor-alpha