Bacterial vaginosis in HIV-infected women induces reversible alterations in the cervical immune environment

J Acquir Immune Defic Syndr. 2008 Dec 15;49(5):520-2. doi: 10.1097/QAI.0b013e318189a7ca.

Abstract

Background: Bacterial vaginosis (BV) has been associated with increased HIV cervicovaginal shedding. We hypothesized that this might relate to BV-associated increases in mucosal activated CD4 T cells, which could enhance local HIV replication.

Methods: Vaginal flora, cytokine/chemokine levels, and mucosal immune cell populations collected by cervical cytobrush were analyzed in 15 HIV-infected Kenyan female sex workers, before and after BV therapy with oral metronidazole.

Results: Therapy reduced the Nugent score in all but 1 participant, and BV elimination was associated with reduced genital levels of interleukin 1beta(IL1beta), interleukin 8 (IL-8), and Regulated Upon Activation Normal T-cell Expressed and Secreted (RANTES). In addition, BV elimination reduced the total number of cervical CD4 T cells, including those expressing the HIV coreceptor CCR5 and the activation marker CD69.

Conclusions: BV induces significant and reversible alterations in cervical immune cell populations and local inflammatory cytokines that would be expected to enhance local HIV replication.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Anti-Bacterial Agents / therapeutic use
  • CD4 Lymphocyte Count
  • Cervix Uteri / cytology
  • Cervix Uteri / immunology*
  • Chemokine CCL5 / analysis
  • Female
  • HIV Infections / complications*
  • Humans
  • Interleukin-1beta / analysis
  • Interleukin-8 / analysis
  • Metronidazole / therapeutic use
  • Vaginosis, Bacterial / complications*
  • Vaginosis, Bacterial / drug therapy
  • Vaginosis, Bacterial / immunology*
  • Virus Shedding / drug effects

Substances

  • Anti-Bacterial Agents
  • Chemokine CCL5
  • Interleukin-1beta
  • Interleukin-8
  • Metronidazole