Obesity and hypertension have differing oxidant handling molecular pathways in age-related chronic kidney disease

Mech Ageing Dev. 2009 Mar;130(3):129-38. doi: 10.1016/j.mad.2008.10.003. Epub 2008 Oct 19.

Abstract

Chronic kidney disease (CKD) in ageing is a burden on health systems worldwide. Rat models of age-related CKD linked with obesity and hypertension were used to investigate alterations in oxidant handling and energy metabolism to identify gene targets or markers for age-related CKD. Young adult (3 months) and old (21-24 months) spontaneously-hypertensive (SHR), normotensive Wistar-Kyoto (WKY) and Wistar rats (normotensive, obese in ageing) were compared for renal functional and physiological parameters, renal fibrosis and inflammation, oxidative stress (hemeoxygenase-1/HO-1), apoptosis and cell injury (including Bax:Bcl-2), phosphorylated and non-phosphorylated forms of oxidant and energy sensing proteins (p66Shc, AMPK), signal transduction proteins (ERK1/2, PKB), and transcription factors (NF-kappaB, FoxO1). All old rats were normoglycemic. Renal fibrosis, tubular epithelial apoptosis, interstitial macrophages and myofibroblasts (all p<0.05), p66Shc/phospho-p66 (p<0.05), Bax/Bcl-2 ratio (p<0.05) and NF-kappaB expression (p<0.01) were highest in old obese Wistars. Expression of phospho-FoxO/FoxO was elevated in old Wistars (p<0.001) and WKYs (p<0.01). SHRs had high levels in young and old rats. Expression of PKB, phospho-PKB, ERK1/2 and phospho-ERK1/2 were significantly elevated in all aged animals. These results suggest that obesity and hypertension have differing oxidant handling and signalling pathways that act in the pathogenesis of age-related CKD.

Publication types

  • Comparative Study
  • Research Support, Non-U.S. Gov't

MeSH terms

  • AMP-Activated Protein Kinases / metabolism
  • Adiposity
  • Age Factors
  • Aging / metabolism*
  • Animals
  • Autophagy
  • Body Weight
  • Chronic Disease
  • Disease Models, Animal
  • Extracellular Signal-Regulated MAP Kinases / metabolism
  • Fibrosis
  • Forkhead Transcription Factors / metabolism
  • Heme Oxygenase (Decyclizing) / metabolism
  • Hypertension / metabolism*
  • Hypertension / pathology
  • Hypertension / physiopathology
  • Kidney / metabolism*
  • Kidney / pathology
  • Kidney / physiopathology
  • Kidney Diseases / metabolism*
  • Kidney Diseases / pathology
  • Kidney Diseases / physiopathology
  • L-Lactate Dehydrogenase / blood
  • Male
  • NF-kappa B / metabolism
  • Nerve Tissue Proteins / metabolism
  • Obesity / metabolism*
  • Obesity / pathology
  • Obesity / physiopathology
  • Oxidants / metabolism*
  • Oxidative Stress*
  • Phosphorylation
  • Proto-Oncogene Proteins c-akt / metabolism
  • Proto-Oncogene Proteins c-bcl-2 / metabolism
  • Rats
  • Rats, Inbred SHR
  • Rats, Inbred WKY
  • Shc Signaling Adaptor Proteins / metabolism
  • Signal Transduction
  • Src Homology 2 Domain-Containing, Transforming Protein 1
  • bcl-2-Associated X Protein / metabolism

Substances

  • Bax protein, rat
  • Forkhead Transcription Factors
  • NF-kappa B
  • Nerve Tissue Proteins
  • Oxidants
  • Proto-Oncogene Proteins c-bcl-2
  • Shc Signaling Adaptor Proteins
  • Shc1 protein, rat
  • Src Homology 2 Domain-Containing, Transforming Protein 1
  • bcl-2-Associated X Protein
  • Foxo1 protein, rat
  • L-Lactate Dehydrogenase
  • Heme Oxygenase (Decyclizing)
  • Hmox1 protein, rat
  • Proto-Oncogene Proteins c-akt
  • Extracellular Signal-Regulated MAP Kinases
  • AMP-Activated Protein Kinases