Anti-interleukin-31-antibodies ameliorate scratching behaviour in NC/Nga mice: a model of atopic dermatitis

Exp Dermatol. 2009 Jan;18(1):35-43. doi: 10.1111/j.1600-0625.2008.00766.x. Epub 2008 Oct 24.

Abstract

Background: Interleukin-31 (IL-31), a novel cytokine, is upregulated in atopic dermatitis skin lesions in humans and skin lesions in the NC/Nga mice, a murine model for atopic dermatitis.

Objective: Here, we investigated the effect of a monoclonal IL-31 antibody on scratching behaviour, weight gain and dermatitis in NC/Nga mice.

Methods: Mice were divided into three groups, n = 10 in each group. Mice were given monoclonal IL-31 rat-anti-mouse antibody 10 mg/kg or albumin intraperitoneally every fifth day for seven weeks. In addition, the mice in one group were not exposed to any form of intervention. The dermatitis score was clinically assessed twice a week. The scratching behaviour was automatically detected and objectively evaluated.

Results: Intervention with IL-31 antibody 10 mg/kg intraperitoneally every fifth day in NC/Nga mice from age 7 weeks reduced the scratching behaviour, but did not have any impact on weight gain or dermatitis.

Conclusion: IL-31 antibody reduces scratching behaviour in an atopic dermatitis-like murine model during the onset of clinical skin manifestations. Our findings suggest IL-31 antibody as a new potential therapeutic approach for pruritus in atopic dermatitis and other pruritic diseases.

MeSH terms

  • Animals
  • Antibodies, Monoclonal / chemistry*
  • Body Weight
  • Dermatitis / immunology*
  • Dermatitis / metabolism
  • Dermatitis, Atopic / immunology
  • Dermatitis, Atopic / metabolism
  • Disease Models, Animal
  • Female
  • Interleukins / chemistry*
  • Interleukins / immunology*
  • Male
  • Mice
  • Pruritus / immunology
  • Pruritus / metabolism
  • Rats
  • Rats, Sprague-Dawley
  • Skin / pathology
  • Skin Diseases / immunology
  • Skin Diseases / metabolism

Substances

  • Antibodies, Monoclonal
  • Interleukins
  • interleukin-31, mouse