Calmodulin- and Ca2(+)-insensitive fatty acid methyltransferase from RINm5F cells. Inhibition by trifluoperazine and W7

Int J Biochem. 1991;23(7-8):769-72. doi: 10.1016/0020-711x(91)90051-n.

Abstract

1. Methylation of endogenous lipids by homogenates of rat insulinoma cells was studied. 2. 3H-methyl groups (38 pmol/mg protein per 10 min) from [3H-methyl]S-adenosyl-L-methionine were incorporated into endogenous lipids, mainly (greater than 80%) into the neutral lipid fraction. 3. The reaction was sensitive to heat, was almost abolished by S-adenosyl-L-homocysteine, but insensitive to the addition of EGTA (5 mM), Ca2+ (5-100 microM) and/or calmodulin (15 microns). 4. At concentrations relevant for calmodulin antagonistic activity strong inhibition by W7 and trifluoperazine (25-100 microM each), but not by CGS 9343B (10 microM), was observed. 5. Calmodulin antagonists of phenothiazine- and sulfonamide-type appear to block the fatty acid methyltransferase in a way unrelated to calmodulin.

MeSH terms

  • Animals
  • Benzimidazoles / pharmacology
  • Calcium / pharmacology*
  • Calmodulin / antagonists & inhibitors
  • Calmodulin / pharmacology*
  • Egtazic Acid / pharmacology
  • Hot Temperature
  • Insulinoma / enzymology*
  • Methylation
  • Methyltransferases / antagonists & inhibitors*
  • Pancreatic Neoplasms / enzymology*
  • Rats
  • S-Adenosylhomocysteine / pharmacology
  • Sulfonamides / pharmacology*
  • Trifluoperazine / pharmacology*
  • Tumor Cells, Cultured

Substances

  • Benzimidazoles
  • Calmodulin
  • Sulfonamides
  • CGS 9343B
  • Trifluoperazine
  • Egtazic Acid
  • W 7
  • S-Adenosylhomocysteine
  • Methyltransferases
  • fatty acid methyltransferase
  • Calcium