Effects of propofol on the mRNA expression of toll-like receptor 4 in BV-2 cells during mimic ischemia-reperfusion injury in vitro

J Huazhong Univ Sci Technolog Med Sci. 2008 Dec;28(6):711-3. doi: 10.1007/s11596-008-0622-7. Epub 2008 Dec 24.

Abstract

This study investigated the effects of propofol on the mRNA expression of Toll-like receptor-4 (TLR4) in BV-2 cells during mimic ischemia-reperfusion (I/R) injury in vitro. BV-2 cells, a mouse microglia line, were cultured and divided into 4 groups at random: control group (group C), ischemia/reperfusion group (group I/R), low-dose propofol (25 micromol/L) intervention group (group PF(25)) and high-dose propofol (100 micromol/L) intervention group (group PF(100)). The mRNA expression of TLR4 and NF-kappaB was measured by means of RT-PCR. TNF-alpha levels in the supernatants of BV-2 cells were detected by ELISA. The results showed that the mRNA expression of TLR4 and NF-kappaB was significantly higher in groups I/R, PF(25) and PF(100) than in group C (P<0.01). And the TNF-alpha level in the supernatants was elevated in groups I/R, PF(25) and PF(100) as compared with that in group C (P<0.01). After pre-treatment with propofol, the mRNA expressions of TLR4 and NF-kappa B and the TNF-alpha level were significantly decreased in groups PF(25) and PF(100) in comparison to those in group I/R (P<0.01). And the decrease in those indicators was more significant in group PF(100) than in group PF(25) (P<0.01). It was concluded that propofol exerted brain-protecting effects during I/R injury by suppressing the mRNA expressions of TLR4 and NF-kappaB and deceasing the TNF-alpha level.

MeSH terms

  • Animals
  • Brain Ischemia / metabolism*
  • Brain Ischemia / pathology
  • Cell Line
  • Mice
  • Microglia / cytology
  • NF-kappa B / genetics
  • NF-kappa B / metabolism
  • Neuroprotective Agents / pharmacology*
  • Propofol / pharmacology*
  • RNA, Messenger / genetics
  • RNA, Messenger / metabolism
  • Random Allocation
  • Reperfusion Injury / metabolism*
  • Reperfusion Injury / pathology
  • Toll-Like Receptor 4 / genetics
  • Toll-Like Receptor 4 / metabolism*
  • Tumor Necrosis Factor-alpha / genetics
  • Tumor Necrosis Factor-alpha / metabolism

Substances

  • NF-kappa B
  • Neuroprotective Agents
  • RNA, Messenger
  • Tlr4 protein, mouse
  • Toll-Like Receptor 4
  • Tumor Necrosis Factor-alpha
  • Propofol