The acute-phase response impairs host defence against Enterococcus faecium peritonitis

Immunology. 2009 Sep;128(1 Suppl):e335-42. doi: 10.1111/j.1365-2567.2008.02967.x. Epub 2008 Oct 30.

Abstract

Enterococcus faecium is an emerging pathogen that causes infections in hospitalized patients with various co-morbid diseases. These underlying diseases are often associated with an acute-phase response that renders patients vulnerable to nosocomial infections. To study the influence of the acute-phase response induced by sterile tissue injury on host defence against E. faecium, mice were injected subcutaneously with either turpentine or casein 1 day before intraperitoneal infection with E. faecium. Control mice were subcutaneously injected with saline or sodium bicarbonate, respectively. Turpentine and casein induced an acute-phase response as reflected by increases in the plasma concentrations of interleukin-6, serum amyloid P and C3. A pre-existent acute-phase response in mice was associated with a strongly reduced capacity to clear E. faecium, resulting in prolonged bacteraemia for several days. The inflammatory response to E. faecium was impaired in mice with an acute-phase response, as shown by reduced capacity to mount a neutrophilic leucocytosis in peripheral blood and by decreased local cytokine concentrations. These data indicate that the acute-phase response impairs host defence against E. faecium, suggesting that this condition may contribute to the increased vulnerability of critically ill patients to enterococcal infections.

MeSH terms

  • Acute-Phase Reaction / chemically induced
  • Acute-Phase Reaction / immunology*
  • Acute-Phase Reaction / metabolism
  • Acute-Phase Reaction / microbiology
  • Animals
  • Caseins / pharmacology
  • Chelating Agents / pharmacology
  • Complement C3 / agonists
  • Complement C3 / immunology
  • Complement C3 / metabolism
  • Cytokines / drug effects
  • Cytokines / immunology
  • Cytokines / metabolism
  • Enterococcus faecium / drug effects
  • Enterococcus faecium / immunology*
  • Female
  • Gram-Positive Bacterial Infections / immunology*
  • Gram-Positive Bacterial Infections / metabolism
  • Gram-Positive Bacterial Infections / microbiology
  • Interleukin-6 / agonists
  • Interleukin-6 / blood
  • Irritants / pharmacology
  • Mice
  • Mice, Inbred C57BL
  • Neutrophils / drug effects
  • Neutrophils / immunology*
  • Neutrophils / metabolism
  • Peritonitis / immunology*
  • Peritonitis / metabolism
  • Peritonitis / microbiology
  • Serum Amyloid P-Component / agonists
  • Serum Amyloid P-Component / immunology
  • Serum Amyloid P-Component / metabolism
  • Turpentine / pharmacology

Substances

  • Caseins
  • Chelating Agents
  • Complement C3
  • Cytokines
  • Interleukin-6
  • Irritants
  • Serum Amyloid P-Component
  • Turpentine