The microglial/macrophagic response at the tumour-brain border of invasive meningiomas

Neuropathol Appl Neurobiol. 2009 Feb;35(1):82-8. doi: 10.1111/j.1365-2990.2008.00960.x.

Abstract

Aims: Little is known about the immune response of the brain to invasive meningiomas. The present study was based upon the hypothesis that the microglial/macrophagic response towards brain-invasive meningiomas is dependent on the intactness of the pial-glial basement membrane.

Methods: We immunostained sections from 40 brain-invasive meningiomas that were graded according to World Health Organization (WHO) 2007 criteria. Thirty-three tumours were histologically WHO grade II (18, 'otherwise benign', and 15, 'otherwise atypical'), and seven, grade III. Microglial/macrophagic cells were labelled with antibodies directed against major histocompatibility complex class II, CD68, CD14 and CD163. Anti-collagen IV was used to visualize basement membranes.

Results: Twenty-five per cent (10/40) meningiomas (1/18 WHO grade II 'otherwise benign', 3/15 grade II 'otherwise atypical' and 6/7 WHO grade III) contained microglial/macrophagic cells at the tumour-brain border. The presence of these cells correlated with the absence of the pial-glial basement membrane (BM) and with WHO grade III. The monocytic response was of two kinds: one consisted of a dense layer of mononuclear cells at the tumour-brain border in nine cases, the other of an elevated number of microglial cells expressing CD14 or CD163 (two cases).

Conclusions: The immune response at the tumour-brain interface correlates with the absence of the pial-glial BM and with malignancy grade. It remains to be established whether the mononuclear cells at the tumour-brain border are native microglia or blood-derived macrophages.

MeSH terms

  • Adult
  • Aged
  • Aged, 80 and over
  • Antigens, CD / metabolism
  • Antigens, Differentiation, Myelomonocytic / metabolism
  • Basement Membrane / immunology
  • Brain / immunology*
  • Brain / metabolism
  • Brain Neoplasms / immunology*
  • Genes, MHC Class II
  • Humans
  • Immunohistochemistry
  • Lipopolysaccharide Receptors / metabolism
  • Macrophages / immunology*
  • Meningioma / immunology*
  • Microglia / immunology*
  • Middle Aged
  • Monocytes / immunology
  • Pia Mater / immunology
  • Receptors, Cell Surface / metabolism

Substances

  • Antigens, CD
  • Antigens, Differentiation, Myelomonocytic
  • CD163 antigen
  • CD68 antigen, human
  • Lipopolysaccharide Receptors
  • Receptors, Cell Surface