Epithelial-specific requirement of FGFR2 signaling during tooth and palate development

J Exp Zool B Mol Dev Evol. 2009 Jun 15;312B(4):343-50. doi: 10.1002/jez.b.21274.

Abstract

Reciprocal interactions between epithelium and mesenchyme are crucial for embryonic development. Fibroblast growth factors (FGFs) are a growth factor family that play an important role in epithelial-mesenchymal tissue interaction. We have generated epithelial-specific conditional knockout mice targeting Fibroblast growth factor receptor 2 (Fgfr2) to investigate the function of FGF signaling during craniofacial development. K14-Cre;Fgfr2(fl/fl) mice have skin defects, retarded tooth formation, and cleft palate. During the formation of the tooth primordium and palatal processes, cell proliferation in the epithelial cells of K14-Cre;Fgfr2(fl/fl) mice is reduced. Thus, FGF signaling via FGFR2 in the epithelium is crucial for cell proliferation activity during tooth and palate development.

Publication types

  • Research Support, N.I.H., Extramural

MeSH terms

  • Animals
  • Cleft Palate / genetics
  • Epithelial Cells / metabolism
  • In Situ Hybridization
  • Mice
  • Palate / growth & development*
  • Receptor, Fibroblast Growth Factor, Type 2 / genetics
  • Receptor, Fibroblast Growth Factor, Type 2 / metabolism*
  • Signal Transduction*
  • Tooth / growth & development*

Substances

  • Receptor, Fibroblast Growth Factor, Type 2