Effects of magnetic nanoparticle of fe(3)o(4) and 5-bromotetrandrine on apoptosis of K562/A02 leukemic cells induced by daunorubicin

Zhongguo Shi Yan Xue Ye Xue Za Zhi. 2009 Feb;17(1):54-9.

Abstract

The aim of this study was to investigate the potential benefit of combination therapy with magnetic nanoparticle of Fe(3)O(4) and 5-Bromotetrandrine (5-BrTet) on chronic leukemia. The apoptosis was detected by flow cytometry (FCM), Wright staining and light microscope; the expressions of BAX and BCL-2 were measured by Western blot. The results showed that combination of daunorubicin (DNR) with either MNP (Fe(3)O(4)) or 5-BrTet exerted a potent cytotoxic effect on K562/A02 cells, while MNP (Fe(3)O(4)) and 5-BrTet co-treatment could synergistically enhance DNR-induced apoptosis. After treated with this regimen, the typical apoptotic morphological features were found in K562/A02 cells; the expression level of BCL-2 decreased and BAX increased markedly. It is concluded that MNP (Fe(3)O(4)) or 5-BrTet with DNR can induce apoptosis in K562/A02 cells, and they show distinct synergism when used together. The down-regulation of BCL-2 and the up-regulation of BAX may play important roles.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Apoptosis / drug effects*
  • Benzylisoquinolines / pharmacology*
  • Daunorubicin / pharmacology
  • Down-Regulation
  • Ferric Compounds / administration & dosage*
  • Gene Expression Regulation, Leukemic
  • Humans
  • K562 Cells
  • Nanoparticles / administration & dosage
  • Proto-Oncogene Proteins c-bcl-2 / metabolism
  • Up-Regulation
  • bcl-2-Associated X Protein / metabolism

Substances

  • BAX protein, human
  • Benzylisoquinolines
  • Ferric Compounds
  • Proto-Oncogene Proteins c-bcl-2
  • bcl-2-Associated X Protein
  • bromotetrandrine
  • Daunorubicin