Akt/protein kinase B overexpression as an accurate prognostic marker in adult diffuse astrocytoma

Acta Neurochir (Wien). 2009 Mar;151(3):263-8; discussion 268. doi: 10.1007/s00701-009-0199-3. Epub 2009 Feb 25.

Abstract

Background: Akt/Protein kinase B (PKB) is a common downstream molecule of Ras signaling essential for cell survival. In an attempt to find a novel prognostic marker of diffuse astrocytoma, we performed an immunohistochemical analysis of Akt/PKB with regard to patient survival and regrowth patterns.

Methods: Twenty-four adult patients with diffuse astrocytoma were similarly managed without early post-operative radiotherapy and followed up for a median period of 7.5 years. They were analysed by immunohistochemistry for Akt/PKB expression as well as p53 protein accumulation, epidermal growth factor receptor (EGFR) expression, and MIB-1 labeling index. The prognostic significance of each molecular covariate was tested by multivariate analysis using Cox's proportional hazard model including age, performance status, and extent of surgical resection.

Findings: Akt/PKB overexpression significantly correlated with both shorter overall survival (OS) and progression-free survival (PFS) (p = 0.0110). All the Akt/PKB-positive patients with post-operative residual tumours experienced tumour recurrences, whereas only a small fraction of the Akt/PKB-negative individuals had recurrences (p = 0.0070). Invasive recurrence into surrounding brain occurred only in the Akt/PKB-overexpressed tumours. In contrast, MIB-1 labeling index correlated only with OS, while p53 protein accumulation correlated only with PFS. The Cox's proportional hazard model identified Akt/PKB overexpression as a significant prognostic factor for shorter PFS (p = 0.0117).

Conclusion: These results show that Akt/PKB overexpression would be suggestive of malignant progression and invasive regrowth of diffuse astrocytoma, and it can serve as a novel prognostic marker for this tumour.

MeSH terms

  • Adult
  • Age Factors
  • Astrocytoma / diagnosis*
  • Astrocytoma / enzymology*
  • Astrocytoma / physiopathology
  • Biomarkers, Tumor / analysis
  • Biomarkers, Tumor / metabolism*
  • Brain Neoplasms / diagnosis*
  • Brain Neoplasms / enzymology*
  • Brain Neoplasms / physiopathology
  • Disease Progression
  • ErbB Receptors / analysis
  • ErbB Receptors / metabolism
  • Female
  • Humans
  • Immunohistochemistry
  • Male
  • Middle Aged
  • Neoplasm Invasiveness / diagnosis
  • Neoplasm Recurrence, Local / diagnosis
  • Neoplasm Recurrence, Local / enzymology
  • Predictive Value of Tests
  • Prognosis
  • Proportional Hazards Models
  • Proto-Oncogene Proteins c-akt / analysis
  • Proto-Oncogene Proteins c-akt / metabolism*
  • Survival Rate
  • Tumor Suppressor Protein p53 / analysis
  • Tumor Suppressor Protein p53 / metabolism
  • Ubiquitin-Protein Ligases / analysis
  • Ubiquitin-Protein Ligases / metabolism

Substances

  • Biomarkers, Tumor
  • Tumor Suppressor Protein p53
  • MIB1 ligase, human
  • Ubiquitin-Protein Ligases
  • EGFR protein, human
  • ErbB Receptors
  • Proto-Oncogene Proteins c-akt