Abstract
Tumorigenesis is regulated by several mechanisms including signalling, transcription and DNA replication. Now a cytoplasmic protein quality-control pathway is implicated in the suppression of breast cancer cell growth, suggesting a new role for quality-control mechanisms in suppressing cells with malignant potential.
MeSH terms
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Animals
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Breast Neoplasms / enzymology
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Breast Neoplasms / genetics
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Breast Neoplasms / pathology*
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Cell Growth Processes
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Female
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Gene Expression Regulation, Neoplastic
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Histone Acetyltransferases / metabolism
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Humans
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Mice
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Mice, Knockout
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Models, Biological
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Nuclear Receptor Coactivator 3
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Trans-Activators / metabolism
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Ubiquitin-Protein Ligases / metabolism*
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Xenograft Model Antitumor Assays
Substances
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Trans-Activators
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Histone Acetyltransferases
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NCOA3 protein, human
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Ncoa3 protein, mouse
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Nuclear Receptor Coactivator 3
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STUB1 protein, human
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Ubiquitin-Protein Ligases