HLA-B27 subtypes and tumor necrosis factor alpha promoter region polymorphism in Iranian patients with ankylosing spondylitis

Eur Cytokine Netw. 2009 Mar;20(1):17-20. doi: 10.1684/ecn.2009.0143.

Abstract

Background: HLA-B27 is an MHC class I molecule that is strongly associated with ankylosing spondylitis (AS). TNF-alpha, as an important cytokine in inflammatory joint disease, might have a role in the process of AS. This study was performed to determine HLA-B27 subtypes among Iranian patients with AS, and to investigate TNF-alpha gene polymorphisms in the patient groups.

Methods: Ninety seven AS patients (74 HLA-B27-positive and 23 HLA-B27-negative) and 137 healthy normal subjects (2 HLA-B27-positive) were enrolled in this study. HLA-B27 positive patients were screened using the polymerase chain reaction, with sequence specific primers (PCR-SSP), for B*27 subtyping. All patients and the controls were also investigated for determination of TNF-alpha polymorphisms using the same method.

Results: Just two subtypes were detected in our patients, namely B*2705 (63.4%) and B*2702 (36.6%). The study of TNF-alpha polymorphisms at position -238 showed that the A allele and AA genotype were significantly over-represented in all patient groups in comparison with the control group (p < 0.001). At position -308, while the A allele and AA genotype were significantly over-represented in the whole patient group (p = 0.01), there was no significant difference between the AS groups and the control group.

Conclusion: It could be suggested that a polymorphism within the TNF-alpha gene at -238 play an important role in AS, although this polymorphism was not related to HLA-B27 subtypes.

MeSH terms

  • Adolescent
  • Adult
  • Alleles
  • Case-Control Studies
  • Child
  • Female
  • Gene Frequency
  • Genotype
  • HLA-B27 Antigen / classification*
  • HLA-B27 Antigen / genetics*
  • Humans
  • Iran
  • Male
  • Middle Aged
  • Polymorphism, Genetic*
  • Promoter Regions, Genetic
  • Spondylitis, Ankylosing / genetics*
  • Spondylitis, Ankylosing / immunology*
  • Tumor Necrosis Factor-alpha / genetics*
  • Young Adult

Substances

  • HLA-B*27:05 antigen
  • HLA-B27 Antigen
  • Tumor Necrosis Factor-alpha