A homozygous MYBPC3 gene mutation associated with a severe phenotype and a high risk of sudden death in a family with hypertrophic cardiomyopathy
Rev Esp Cardiol. 2009 May;62(5):572-5.
doi: 10.1016/s1885-5857(09)71841-9.
[Article in
English,
Spanish]
Affiliation
- 1 Servicio de Cardiología, Complejo Hospitalario Universitario A Coruña, A Coruña, Spain.
Abstract
Genetic studies can play a key role in the comprehensive evaluation of familiar hypertrophic cardiomyopathy and in the development of individualized medicine. Although only a few cases have been described, there exists a group of patients with complex genotypes that are associated with severe disease manifestations and a high risk of sudden death. We describe a family in which some members experienced the early development of systolic and diastolic dysfunction while others experienced sudden death at a young age. We identified a novel homozygous mutation (IVS6+5G>A) in the myosin-binding protein-C gene that explained the phenotype of affected individuals and that enabled us to estimate the risk in other family members and to offer genetic counseling.
Publication types
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Case Reports
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Research Support, Non-U.S. Gov't
MeSH terms
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Adult
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Aged
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Cardiomyopathy, Hypertrophic / diagnostic imaging
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Cardiomyopathy, Hypertrophic / genetics*
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Carrier Proteins / genetics*
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Death, Sudden, Cardiac / epidemiology*
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Female
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Humans
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Male
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Middle Aged
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Mutation / genetics*
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Mutation / physiology*
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Pedigree
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Phenotype
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Polymorphism, Single-Stranded Conformational
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Risk
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Ultrasonography
Substances
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Carrier Proteins
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myosin-binding protein C