Reversal of cardiac dysfunction after enzyme replacement in patients with infantile-onset Pompe disease

J Pediatr. 2009 Aug;155(2):271-5.e2. doi: 10.1016/j.jpeds.2009.03.015. Epub 2009 May 31.

Abstract

Objective: To compare the effects of enzyme replacement therapy (ERT) on cardiac performance in symptomatic and symptom-free infants with Pompe disease.

Study design: Patients diagnosed between 1983 and 2008 were identified. Before the initiation of ERT, systolic dysfunction appeared only in patients > or = 5 months; thus we used this cut-point in age to divide clinically symptomatic patients into early and late treatment groups (Clin-E and Clin-L). Newborn screening (NBS) identified symptom-free patients.

Results: Among a total of 40 patients, 14 received ERT: 5 in the Clin-L, 4 in the Clin-E, and 5 in the NBS groups. All patients showed cardiomegaly, hypertrophic myocardium, and elevated B-type natriuretic peptide (measured in the Clin-E and NBS groups). ERT improved the survival and outcomes. Regressed myocardial hypertrophy and lowered B-type natriuretic peptide level occurred after 1 to 6 months of ERT. Nonetheless, there were 2 deaths and 2 survivors requiring ventilator support in the Clin-L group. Despite the regressed QRS voltage and shortened QT dispersion, life-threatening arrhythmias were still observed in 3, but none in the NBS group.

Conclusion: ERT may restore the cardiac function in both symptomatic and symptom-free patients, but the beneficial effect may be unpredictable if given after the age of 5 months.

MeSH terms

  • Cardiomegaly / drug therapy*
  • Cardiomegaly / etiology
  • Electrocardiography
  • Glycogen Storage Disease Type II / blood
  • Glycogen Storage Disease Type II / complications
  • Glycogen Storage Disease Type II / drug therapy*
  • Humans
  • Hypertrophy, Left Ventricular / drug therapy*
  • Hypertrophy, Left Ventricular / etiology
  • Infant
  • Natriuretic Peptide, Brain / blood
  • Prospective Studies
  • Stroke Volume / drug effects*
  • Tachycardia / etiology
  • alpha-Glucosidases / therapeutic use*

Substances

  • Natriuretic Peptide, Brain
  • alpha-Glucosidases