Evidence that insulin activates casein kinase 2 in rat epididymal fat-cells and that this may result in the increased phosphorylation of an acid-soluble 22 kDa protein

Biochem J. 1991 Oct 15;279 ( Pt 2)(Pt 2):545-51. doi: 10.1042/bj2790545.

Abstract

Casein kinase 2 activity as measured by phosphorylation of the peptide substrate Arg-Arg-Arg-Glu-Glu-Glu-Thr-Glu-Glu-Glu is increased by about 50% in extracts from insulin-treated epididymal fat-pads or isolated fat-cells after purification by Mono Q chromatography. Insulin acts to increase the Vmax. of the kinase. An acid-soluble protein with an apparent subunit molecular mass of about 22 kDa appears to be a substrate for casein kinase 2. The protein possesses a number of properties in common with the acid-soluble heat-stable 22 kDa protein which exhibits increased phosphorylation in rat adipose tissue exposed to insulin.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Adipose Tissue / enzymology*
  • Amino Acid Sequence
  • Animals
  • Casein Kinases
  • Enzyme Activation / drug effects
  • Epididymis
  • Insulin / pharmacology*
  • Male
  • Molecular Sequence Data
  • Molecular Weight
  • Peptide Mapping
  • Phosphoproteins / chemistry
  • Phosphoproteins / metabolism*
  • Phosphorylation
  • Protein Kinases / metabolism*
  • Rats
  • Rats, Inbred Strains

Substances

  • Insulin
  • Phosphoproteins
  • Protein Kinases
  • Casein Kinases