MicroRNA 92b controls the G1/S checkpoint gene p57 in human embryonic stem cells

Stem Cells. 2009 Jul;27(7):1524-8. doi: 10.1002/stem.84.

Abstract

Human embryonic stem (ES) cells exhibit a shorter G(1) cell cycle phase than most somatic cells. Here, we examine the role of an abundant, human ES cell-enriched microRNA, miR-92b, in cell cycle distribution. Inhibition of miR-92b in human ES cells results in a greater number of cells in the G(1) phase and a lower number in the S phase. Conversely, overexpression of miR-92b in differentiated cells results in a decreased number of cells in G1 phase and an increased number in S-phase. p57, a gene whose product inhibits G(1) to S-phase progression, is one of the predicted targets of miR-92b. Inhibition of miR-92b in human ES cells increases p57 protein levels, and miR-92b overexpression in differentiated cells decreases p57 protein levels. Furthermore, miR-92b inhibits a luciferase reporter construct that includes part of the 3' untranslated region of the p57 gene containing the predicted target of the miR-92b seed sequence. Thus, we show that the miRNA miR-92b directly downregulates protein levels of the G(1)/S checkpoint gene p57. STEM CELLS 2009;27:1524-1528.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Blotting, Western
  • Cell Cycle / genetics
  • Cell Cycle Proteins / genetics
  • Cell Cycle Proteins / metabolism
  • Cyclin-Dependent Kinase Inhibitor p57 / genetics
  • Cyclin-Dependent Kinase Inhibitor p57 / metabolism*
  • Embryonic Stem Cells / cytology*
  • Embryonic Stem Cells / metabolism*
  • Flow Cytometry
  • G1 Phase / genetics*
  • Humans
  • MicroRNAs / genetics
  • MicroRNAs / physiology*
  • Models, Genetic
  • S Phase / genetics*

Substances

  • CDKN1C protein, human
  • Cell Cycle Proteins
  • Cyclin-Dependent Kinase Inhibitor p57
  • MicroRNAs