Drug resistant ADLTE and recurrent partial status epilepticus with dysphasic features in a family with a novel LGI1mutation: electroclinical, genetic, and EEG/fMRI findings

Epilepsia. 2009 Nov;50(11):2481-6. doi: 10.1111/j.1528-1167.2009.02181.x. Epub 2009 Jun 22.

Abstract

Purpose: We characterized a family with autosomal dominant lateral temporal epilepsy (ADLTE) whose proband presented uncommon electroclinical findings such as drug-resistant seizures and recurrent episodes of status epilepticus with dysphasic features.

Methods: The electroclinical characteristics and LGI1 genotype were defined in the family. In the proband, the ictal pattern was documented during video-EEG monitoring and epileptic activity was mapped by EEG/fMRI.

Results: The affected members who were studied had drug-resistant seizures. In the proband, seizures with predominant dysphasic features often occurred as partial status epilepticus. The video-EEG-documented ictal activity and fMRI activation clearly indicated the elective involvement of the left posterior lateral temporal cortex. Sequencing of LGI1 exons revealed a heterozygous c.367G>A mutation in exon 4, resulting in a Glu123Lys substitution in the protein sequence.

Conclusions: The uncommon clinical pattern (high seizure frequency, drug-resistance) highlights the variability of the ADLTE phenotype and extends our knowledge of the clinical spectrum associated with LGI1 mutations.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Adult
  • Aged
  • Anticonvulsants / therapeutic use
  • DNA Mutational Analysis
  • Drug Resistance
  • Electroencephalography / statistics & numerical data*
  • Epilepsy, Temporal Lobe / drug therapy
  • Epilepsy, Temporal Lobe / genetics*
  • Epilepsy, Temporal Lobe / physiopathology*
  • Exons / genetics
  • Family
  • Female
  • Genetic Linkage / genetics
  • Genotype
  • Humans
  • Intracellular Signaling Peptides and Proteins
  • Magnetic Resonance Imaging / statistics & numerical data*
  • Male
  • Pedigree
  • Phenotype
  • Point Mutation / genetics*
  • Proteins / genetics*
  • Status Epilepticus / genetics
  • Status Epilepticus / physiopathology
  • Temporal Lobe / physiopathology
  • Video Recording

Substances

  • Anticonvulsants
  • Intracellular Signaling Peptides and Proteins
  • LGI1 protein, human
  • Proteins