Gene expression of the invasive phenotype of TNF-alpha-treated MCF-7 cells

Biomed Pharmacother. 2009 Jul;63(6):421-8. doi: 10.1016/j.biopha.2009.04.032. Epub 2009 Jun 13.

Abstract

Background and objectives: TNF-alpha secreted by tumor cells and macrophages that have infiltrated into the micro-environment of a tumor may promote the metastasis of a variety of malignant cancers, including breast cancer. The present study was designed to detect gene expression changes in metastatic MCF-7 cells treated with a low dose of TNF-alpha (20ng/mL), and to further explore the mechanisms by which TNF-alpha can contribute to metastasis.

Methods: Transwell assays were performed to evaluate the invasive phenotype of MCF-7 cells. Samples for cDNA array analysis were collected 3h and 24h after pre-treatment with TNF-alpha and changes in gene expression were quantitated.

Results: The invasive phenotype of MCF-7 cells was enhanced by the exposure of MCF-7 cells to a low dose of TNF-alpha. Gene expression profiles of 39 genes significantly increased or decreased after treatment with TNF-alpha, 6 of which were genes not previously associated with regulation by TNF-alpha. Genes to promote metastasis, as well as to inhibit metastasis, were identified with some changes being time dependent.

Conclusions: TNF-alpha can enhance the invasive capacity of MCF-7 cells by affecting the expression of a group of genes that have roles in various steps of metastasis. Mechanistic insights into the role of TNF-alpha in tumor cell metastasis are discussed.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Breast Neoplasms / genetics*
  • Cell Line, Tumor
  • Female
  • Gene Expression Profiling / methods
  • Gene Expression Regulation, Neoplastic / drug effects*
  • Humans
  • Neoplasm Metastasis / genetics
  • Oligonucleotide Array Sequence Analysis
  • Phenotype*
  • Time Factors
  • Tumor Necrosis Factor-alpha / metabolism
  • Tumor Necrosis Factor-alpha / pharmacology*

Substances

  • Tumor Necrosis Factor-alpha