Regulation of bone formation and remodeling by G-protein-coupled receptor 48

Development. 2009 Aug;136(16):2747-56. doi: 10.1242/dev.033571. Epub 2009 Jul 15.

Abstract

G-protein-coupled receptor (GPCR) 48 (Gpr48; Lgr4), a newly discovered member of the glycoprotein hormone receptor subfamily of GPCRs, is an orphan GPCR of unknown function. Using a knockout mouse model, we have characterized the essential roles of Gpr48 in bone formation and remodeling. Deletion of Gpr48 in mice results in a dramatic delay in osteoblast differentiation and mineralization, but not in chondrocyte proliferation and maturation, during embryonic bone formation. Postnatal bone remodeling is also significantly affected in Gpr48(-/-) mice, including the kinetic indices of bone formation rate, bone mineral density and osteoid formation, whereas the activity and number of osteoclasts are increased as assessed by tartrate-resistant acid phosphatase staining. Examination of the molecular mechanism of Gpr48 action in bone formation revealed that Gpr48 can activate the cAMP-PKA-CREB signaling pathway to regulate the expression level of Atf4 in osteoblasts. Furthermore, we show that Gpr48 significantly downregulates the expression levels of Atf4 target genes/proteins, such as osteocalcin (Ocn; Bglap2), bone sialoprotein (Bsp; Ibsp) and collagen. Together, our data demonstrate that Gpr48 regulates bone formation and remodeling through the cAMP-PKA-Atf4 signaling pathway.

Publication types

  • Research Support, N.I.H., Extramural
  • Research Support, Non-U.S. Gov't

MeSH terms

  • Activating Transcription Factor 4 / genetics
  • Activating Transcription Factor 4 / metabolism
  • Animals
  • Bone Density
  • Bone Remodeling / physiology*
  • Bone and Bones / cytology
  • Bone and Bones / physiology
  • Cell Differentiation / physiology
  • Cells, Cultured
  • Chondrocytes / cytology
  • Chondrocytes / metabolism
  • Collagen / genetics
  • Collagen / metabolism
  • Cyclic AMP / metabolism
  • Cyclic AMP Response Element-Binding Protein / metabolism
  • Cyclic AMP-Dependent Protein Kinases / metabolism
  • Embryo, Mammalian / anatomy & histology
  • Embryo, Mammalian / physiology
  • Gene Expression Regulation, Developmental
  • Integrin-Binding Sialoprotein
  • Mice
  • Mice, Knockout
  • Osteoblasts / cytology
  • Osteoblasts / physiology
  • Osteocalcin / metabolism
  • Osteoclasts / cytology
  • Osteoclasts / metabolism
  • Osteogenesis / physiology*
  • Osteoporosis / physiopathology
  • Protein Isoforms / genetics
  • Protein Isoforms / metabolism*
  • Receptors, G-Protein-Coupled / genetics
  • Receptors, G-Protein-Coupled / metabolism*
  • Sialoglycoproteins / metabolism
  • Signal Transduction / physiology

Substances

  • Atf4 protein, mouse
  • Creb1 protein, mouse
  • Cyclic AMP Response Element-Binding Protein
  • Ibsp protein, mouse
  • Integrin-Binding Sialoprotein
  • LGR4 protein, mouse
  • Protein Isoforms
  • Receptors, G-Protein-Coupled
  • Sialoglycoproteins
  • Osteocalcin
  • Activating Transcription Factor 4
  • Collagen
  • Cyclic AMP
  • Cyclic AMP-Dependent Protein Kinases