Fetal-maternal HLA-C mismatch is associated with decidual T cell activation and induction of functional T regulatory cells

J Reprod Immunol. 2009 Nov;82(2):148-57. doi: 10.1016/j.jri.2009.05.003. Epub 2009 Jul 23.

Abstract

Human leukocyte antigen-C (HLA-C) is the only polymorphic classical histocompatibility antigen expressed by fetal trophoblasts at the fetal-maternal interface. Interactions between HLA-C and decidual natural killer (NK) cells may facilitate trophoblast invasion into maternal tissue. Thus far no evidence has been provided that decidual T cells specifically recognize and respond to fetal alloantigens at the fetal-maternal interface. In this study, we show that pregnancies containing a HLA-C mismatched child induce an increased percentage of CD4(+)CD25(dim) activated T cells in decidual tissue. In addition, HLA-C mismatched pregnancies exhibit a decidual lymphocyte response to fetal cells and contain functional CD4(+)CD25(bright) regulatory T cells in decidual tissue, whereas HLA-C matched pregnancies do not. This suggests that decidual T cells specifically recognize fetal HLA-C at the fetal-maternal interface but are prevented from inducing a destructive immune response in uncomplicated pregnancies.

MeSH terms

  • Adult
  • CD4 Antigens / biosynthesis
  • Cell Count
  • Cell Proliferation
  • Decidua / immunology
  • Decidua / metabolism*
  • Decidua / pathology
  • Female
  • HLA-C Antigens / immunology
  • HLA-C Antigens / metabolism*
  • Histocompatibility Testing
  • Histocompatibility, Maternal-Fetal*
  • Humans
  • Immune Tolerance
  • Interleukin-2 Receptor alpha Subunit / biosynthesis
  • Isoantigens / immunology
  • Lymphocyte Activation
  • Pregnancy
  • T-Cell Antigen Receptor Specificity / immunology
  • T-Lymphocyte Subsets / immunology
  • T-Lymphocyte Subsets / metabolism*
  • T-Lymphocyte Subsets / pathology
  • T-Lymphocytes, Regulatory / immunology
  • T-Lymphocytes, Regulatory / metabolism*
  • T-Lymphocytes, Regulatory / pathology

Substances

  • CD4 Antigens
  • HLA-C Antigens
  • Interleukin-2 Receptor alpha Subunit
  • Isoantigens