Cyclooxygenase-2 (COX-2) G-765C is a protective factor for coronary artery disease but not for ischemic stroke: a meta-analysis

Atherosclerosis. 2009 Dec;207(2):492-5. doi: 10.1016/j.atherosclerosis.2009.06.029. Epub 2009 Jul 8.

Abstract

Objective: Previous case-control studies suggested the single nucleotide polymorphism of cyclooxygenase-2 (COX-2) gene (G-765C) is associated with coronary artery disease (CAD) and ischemic stroke. However, other studies did not confirm this relationship. The objective was to assess the relationship of COX-2 G-765C and CAD and ischemic stroke, using a meta-analysis.

Methods: Databases, including PubMed, EMbase, CBM and CNKI, were searched to get the genetic association studies. Data were extracted by two authors and pooled odds ratio (OR) with 95% confidence interval (CI) was calculated.

Results: The meta-analysis included 4930 CAD patients and 17,997 controls, as well as 1628 ischemic stroke patients and 17,653 controls. For CAD, the pooled OR of -765C was 0.80 (95%CI: 0.65-0.98) compared to wild type allele in recessive model, and was 0.99 (95%CI: 0.92-1.06) in dominant model. For ischemic stroke, the pooled OR of -765C was 1.11 (95%CI: 0.88-1.42) in recessive model compared to wild type allele, and was 1.05 (95%CI: 0.93-1.18) in dominant model. No publication bias was found in this meta-analysis.

Conclusion: The synthesis of available evidence supports that the COX-2 G-765C is protective for CAD. However, it is not associated with ischemic stroke.

Publication types

  • Meta-Analysis
  • Research Support, Non-U.S. Gov't

MeSH terms

  • Brain Ischemia / enzymology
  • Brain Ischemia / genetics*
  • Case-Control Studies
  • Coronary Disease / enzymology
  • Coronary Disease / genetics*
  • Coronary Disease / prevention & control
  • Cyclooxygenase 2 / genetics*
  • Evidence-Based Medicine
  • Gene Frequency
  • Genetic Association Studies
  • Genetic Predisposition to Disease
  • Humans
  • Odds Ratio
  • Polymorphism, Single Nucleotide*
  • Risk Assessment
  • Risk Factors
  • Stroke / enzymology
  • Stroke / genetics*

Substances

  • Cyclooxygenase 2
  • PTGS2 protein, human