Efficient and stable transduction of resting B lymphocytes and primary chronic lymphocyte leukemia cells using measles virus gp displaying lentiviral vectors

Blood. 2009 Oct 8;114(15):3173-80. doi: 10.1182/blood-2009-05-220798. Epub 2009 Aug 10.

Abstract

Up to now, no lentiviral vector (LV) tool existed to govern efficient and stable gene delivery into quiescent B lymphocytes, which hampers its application in gene therapy and immunotherapy areas. Here, we report that LVs incorporating measles virus (MV) glycoproteins, H and F, on their surface allowed transduction of 50% of quiescent B cells, which are not permissive to VSVG-LV transduction. This high transduction level correlated with B-cell SLAM expression and was not at cost of cell-cycle entry or B-cell activation. Moreover, the naive and memory phenotypes of transduced resting B cells were maintained. Importantly, H/F-LVs represent the first tool permitting stable transduction of leukemic cancer cells, B-cell chronic lymphocytic leukemia cells, blocked in G(0)/G(1) early phase of the cell cycle. Thus, H/F-LV transduction overcomes the limitations of current LVs by making B cell-based gene therapy and immunotherapy applications feasible. These new LVs will facilitate antibody production and the study of gene functions in these healthy and cancer immune cells.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Antigens, CD / biosynthesis
  • B-Lymphocytes / metabolism*
  • B-Lymphocytes / pathology
  • Cell Line
  • Female
  • G1 Phase
  • Gene Expression Regulation
  • Genetic Therapy / methods
  • Genetic Vectors*
  • Glycoproteins / genetics
  • Glycoproteins / metabolism*
  • HIV-1*
  • Humans
  • Leukemia, Lymphocytic, Chronic, B-Cell
  • Lymphocyte Activation
  • Male
  • Measles virus*
  • Receptors, Cell Surface / biosynthesis
  • Resting Phase, Cell Cycle
  • Signaling Lymphocytic Activation Molecule Family Member 1
  • Transduction, Genetic / methods*
  • Viral Fusion Proteins / genetics
  • Viral Fusion Proteins / metabolism*

Substances

  • Antigens, CD
  • Glycoproteins
  • Receptors, Cell Surface
  • Viral Fusion Proteins
  • Signaling Lymphocytic Activation Molecule Family Member 1