Linking metastasis suppression with metastamiR regulation

Cell Cycle. 2009 Sep 1;8(17):2673-5. doi: 10.4161/cc.8.17.9303. Epub 2009 Sep 23.

Abstract

Cancer metastasis requires the coordinate expression of multiple genes during every step of the metastatic cascade. Molecules that regulate these genetic programs have the potential to impact metastasis at multiple levels. Breast cancer metastasis suppressor 1 (BRMS1) suppresses metastasis by inhibiting multiple steps in the cascade through regulation of many protein-encoding, metastasis-associated genes as well as metastasis-regulatory microRNA, termed metastamiR. In this Feature, we will highlight connections between BRMS1 biology and regulation of metastamiR.

Publication types

  • Editorial
  • Research Support, N.I.H., Extramural
  • Research Support, Non-U.S. Gov't
  • Research Support, U.S. Gov't, Non-P.H.S.

MeSH terms

  • Gene Expression Regulation, Neoplastic
  • Histone Deacetylases / metabolism
  • Humans
  • MicroRNAs / metabolism*
  • Neoplasm Metastasis*
  • Neoplasm Proteins / metabolism*
  • Repressor Proteins
  • Sin3 Histone Deacetylase and Corepressor Complex / metabolism

Substances

  • BRMS1 protein, human
  • MicroRNAs
  • Neoplasm Proteins
  • Repressor Proteins
  • Histone Deacetylases
  • Sin3 Histone Deacetylase and Corepressor Complex