Pentoxifylline attenuates leukoreduced stored blood-induced neutrophil activation through inhibition of mitogen-activated protein kinases

Immunopharmacol Immunotoxicol. 2010 Mar;32(1):74-81. doi: 10.1080/08923970903143965.

Abstract

Context: Transfusion of packed red blood cells is an independent risk factor for postoperative bacterial infections and multiple organ failure.

Materials and methods: Whole blood was obtained from healthy volunteers to investigate the role of mitogen associated protein kinase (MAPK) pathways in PRBC-induced neutrophil respiratory burst, hypothesizing that the attenuating effects of pentoxifylline on this process are due to modulations in MAPK-associated signaling.

Results: Pre-incubation of neutrophils with supernatant prior to fMLP stimulation increased p38 MAPK and ERK phosphorylation over fMLP alone pentoxifylline significantly reduced p38MAPK and ERK phosphorylation in similarly stimulated neutrophils.

Conclusion: The addition of pentoxifylline stored red blood cells attenuates neutrophil activation.

MeSH terms

  • Erythrocytes / physiology*
  • Extracellular Signal-Regulated MAP Kinases / antagonists & inhibitors*
  • Humans
  • Leukocyte Reduction Procedures
  • Neutrophil Activation / drug effects*
  • Pentoxifylline / pharmacology*
  • Phosphodiesterase Inhibitors / pharmacology*
  • Phosphorylation
  • Respiratory Burst / drug effects
  • p38 Mitogen-Activated Protein Kinases / antagonists & inhibitors*

Substances

  • Phosphodiesterase Inhibitors
  • Extracellular Signal-Regulated MAP Kinases
  • p38 Mitogen-Activated Protein Kinases
  • Pentoxifylline