The role of steroid receptor coactivator-3 (SRC-3) in human malignant disease

Eur J Surg Oncol. 2010 Mar;36(3):224-9. doi: 10.1016/j.ejso.2009.08.002. Epub 2009 Aug 28.

Abstract

Background: The p160 steroid receptor coactivator (SRC) family is critical to the transcriptional activation function of nuclear hormone receptors. A key member of this family is SRC-3, initially found to be amplified and expressed in breast cancer it has subsequent been shown to be expressed in malignant disease arising from a wide range of other organs. An understanding of the potential role of SRC-3 in the pathogenesis and its possible prognostic role in a broad range of tumours will improve our general understanding of carcinogenesis as well as potentially leading to a new prognostic marker as well as new therapeutic targets.

Methods: Relevant papers were identified by searching the PubMed and MEDLINE databases for article published until 28th February 2009. Only articles published in English were considered. The search terms included "SRC-3", "AIB1" in association with the following terms: "human", "cancer" and "malignant disease". The search focused on malignant disease arising outside of the mammary gland. Full articles were obtained and references were checked for additional material when appropriate.

Results: SRC-3 is amplified and expressed in a wide spectrum of human malignant diseases and appears to be a potential prognostic marker in a number of different tumours.

Conclusion: SRC-3 appears to be implicated in the possible risk of developing prostate and ovarian cancer. Its presence appears to be a marker of aggressive disease. Further research is required to determine its predictive and prognostic utility given the relative paucity of studies for each specific malignant disease.

Publication types

  • Research Support, Non-U.S. Gov't
  • Review

MeSH terms

  • Animals
  • Biomarkers, Tumor / genetics
  • Biomarkers, Tumor / metabolism
  • DNA, Plant / genetics*
  • Gene Expression Regulation, Neoplastic*
  • Genetic Predisposition to Disease
  • Humans
  • Neoplasms / genetics*
  • Neoplasms / metabolism
  • Nuclear Receptor Coactivator 3 / genetics*
  • Nuclear Receptor Coactivator 3 / metabolism
  • Prognosis
  • Transcription, Genetic / genetics*

Substances

  • Biomarkers, Tumor
  • DNA, Plant
  • NCOA3 protein, human
  • Nuclear Receptor Coactivator 3