Arecoline, but not haloperidol, induces changes in the permeability of the blood-brain barrier in the rat

J Pharm Pharmacol. 1990 Feb;42(2):135-8. doi: 10.1111/j.2042-7158.1990.tb05370.x.

Abstract

The aim of the present study was to investigate the existence of alterations of the blood-brain barrier (BBB) permeability in rats injected with centrally acting drugs, by calculating a unidirectional blood-to-brain transfer constant (Ki) for the circulating tracer [14C]-alpha-aminoisobutyric acid. The intraperitoneal (i.p.) injection of the dopaminergic antagonist haloperidol (1 mg kg-1) did not modify the regional BBB permeability. When the cholinomimetic agent arecoline hydrobromide (6.25 mg kg-1) was injected i.p. into methylatropine-pretreated rats, it induced a significant decrease of Ki values within the frontal cortex, parietal cortex, striatum and brain-stem. Our findings emphasize two concepts: (1) centrally acting drugs, such as arecoline, can induce changes in the BBB permeability, through several mechanisms; (2) there is no predictable correlation of drug stimulation of specific brain neuronal pathways and changes in the permeability of the BBB.

Publication types

  • Comparative Study

MeSH terms

  • Aminoisobutyric Acids / pharmacology
  • Animals
  • Arecoline / pharmacology*
  • Atropine / pharmacology
  • Blood Volume / drug effects
  • Blood-Brain Barrier / drug effects*
  • Haloperidol / pharmacology*
  • Kinetics
  • Male
  • Permeability
  • Rats
  • Rats, Inbred Strains
  • Regional Blood Flow / drug effects

Substances

  • Aminoisobutyric Acids
  • 2-aminoisobutyric acid
  • Arecoline
  • Atropine
  • Haloperidol