Abstract
The potential role of cancer stem-like cells (CSCs) in chemoresistance of human oral squamous cell carcinoma (OSCC) was examined. A small subpopulation (1-2%) of CD133(+) CSCs was identified in OSCC cell lines and tissues. These CD133(+) CSCs possess higher clonogenicity, invasiveness, and increased in vivo tumorigenicity as compared to CD133(-) counterparts. Meanwhile, CD133(+) CSCs were substantially resistant to standard chemotherapy, wherein both in vitro and in vivo treatment with paclitaxel resulted in a marked enrichment for CD133(+) CSCs. Our data suggest that CD133(+) cells represent a small subpopulation of CSCs that may contribute to chemoresistance in human OSCC.
Copyright 2009 Elsevier Ireland Ltd. All rights reserved.
Publication types
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Research Support, N.I.H., Extramural
MeSH terms
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AC133 Antigen
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Animals
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Antigens, CD / metabolism*
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Antineoplastic Agents / adverse effects*
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Carcinoma, Squamous Cell / drug therapy
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Carcinoma, Squamous Cell / pathology*
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Colony-Forming Units Assay
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Drug Resistance, Neoplasm*
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Female
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Flow Cytometry
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Glycoproteins / metabolism*
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Humans
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Immunoenzyme Techniques
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Mice
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Mice, Nude
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Mouth Neoplasms / drug therapy
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Mouth Neoplasms / pathology*
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Neoplastic Stem Cells / metabolism
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Neoplastic Stem Cells / pathology*
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Peptides / metabolism*
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RNA, Messenger / genetics
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RNA, Messenger / metabolism
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Reverse Transcriptase Polymerase Chain Reaction
Substances
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AC133 Antigen
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Antigens, CD
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Antineoplastic Agents
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Glycoproteins
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PROM1 protein, human
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Peptides
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Prom1 protein, mouse
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RNA, Messenger