Genetically engineered S and F1C fimbriae differ in their contribution to adherence of Escherichia coli to cultured renal tubular cells

Infect Immun. 1990 Oct;58(10):3434-7. doi: 10.1128/iai.58.10.3434-3437.1990.

Abstract

Escherichia coli K-12 strains producing S-fimbrial adhesins, F1C fimbriae, and mutagenized fimbriae were tested in a binding assay with a renal tubular cell line. S-fimbrial adhesins and F1C fimbriae mediated binding to tubular cells. The SfaA, SfaG, and SfaS subunits of S fimbriae contributed to attachment. Site-specific mutations in the sfaS gene reduced binding. The inhibition profile of F1C fimbriae resembled that of S fimbriae.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Adhesins, Escherichia coli
  • Animals
  • Bacterial Adhesion*
  • Bacterial Outer Membrane Proteins / genetics
  • Cell Line
  • Escherichia coli / genetics
  • Escherichia coli / physiology*
  • Fimbriae, Bacterial / immunology
  • Fimbriae, Bacterial / physiology*
  • Genes, Bacterial
  • Kidney Tubules / microbiology*
  • Recombinant Fusion Proteins
  • Swine

Substances

  • Adhesins, Escherichia coli
  • Bacterial Outer Membrane Proteins
  • Recombinant Fusion Proteins