Abstract
Antigen-specific gamma interferon (IFN-gamma) has been demonstrated to participate in protection against Bordetella pertussis infection. Circulating mononuclear cells from B. pertussis-infected and from pertussis-vaccinated infants secrete high amounts of IFN-gamma after in vitro stimulation by B. pertussis antigens, but with a large variation in the secreted IFN-gamma levels between individuals. We show here that the inhibition of the specific IFN-gamma response can be at least partially attributed to IL-10 secretion by monocytes. This IL-10 secretion was not associated with polymorphisms at positions -1082, -819, and -592 of the IL-10 gene promoter, suggesting that other genetic or environmental factors affect IL-10 expression and secretion.
Publication types
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Research Support, Non-U.S. Gov't
MeSH terms
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Adhesins, Bacterial / pharmacology
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Alleles
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Antibodies, Monoclonal / pharmacology
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Bordetella pertussis / immunology*
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Genotype
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Humans
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Immunologic Factors / pharmacology
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Infant
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Interferon-gamma / agonists
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Interferon-gamma / biosynthesis
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Interferon-gamma / immunology
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Interleukin-10 / antagonists & inhibitors
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Interleukin-10 / biosynthesis
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Interleukin-10 / genetics
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Interleukin-10 / immunology*
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Interleukin-12 / agonists
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Interleukin-12 / biosynthesis
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Interleukin-12 / immunology
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Leukocytes, Mononuclear / drug effects
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Leukocytes, Mononuclear / immunology*
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Leukocytes, Mononuclear / metabolism
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Leukocytes, Mononuclear / microbiology
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Longitudinal Studies
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Pertussis Toxin / pharmacology
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Pertussis Vaccine / immunology*
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Polymorphism, Genetic / genetics
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Polymorphism, Genetic / immunology
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Virulence Factors, Bordetella / pharmacology
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Whooping Cough / immunology*
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Whooping Cough / microbiology
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Whooping Cough / prevention & control
Substances
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Adhesins, Bacterial
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Antibodies, Monoclonal
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Immunologic Factors
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Pertussis Vaccine
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Virulence Factors, Bordetella
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filamentous hemagglutinin adhesin, Bordetella pertussis
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Interleukin-10
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Interleukin-12
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Interferon-gamma
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Pertussis Toxin