Lumenal part of the DC-LAMP protein is not required for induction of antigen-specific T cell responses by means of antigen-DC-LAMP messenger RNA-electroporated dendritic cells

Hum Gene Ther. 2010 Apr;21(4):479-85. doi: 10.1089/hum.2009.080.

Abstract

Previous studies showed that stimulation of T cells derived from HIV-1-infected patients with autologous dendritic cells electroporated with mRNA encoding HIV antigens can induce antigen-specific T cell responses in vitro. Linking the antigen to an MHC class II-targeting sequence, such as dendritic cell lysosome-associated membrane protein (DC-LAMP), in the mRNA construct results in presentation of antigenic peptides in both MHC class I and class II molecules and therefore enhances the induced T cell responses. To analyze whether the lumenal domain of DC-LAMP is required for optimal induction of cellular immunity against HIV antigens, we compared fusion constructs with or without the lumenal domain of the DC-LAMP protein. A human codon-optimized consensus Gag sequence and a chimeric cDNA sequence encompassing Tat, Rev, and Nef codons (TaReNef ) were cloned into a vector containing the DC-LAMP sequence with or without its lumenal domain. The Gag protein lacking the DC-LAMP-derived sequence altogether elicited only weak T cell responses. DCs electroporated with Gag or TaReNef linked to DC-LAMP were able to elicit similar levels of antigen-specific CD4(+) and CD8(+) T cell responses for both Gag and TaReNef, irrespective of the addition of the DC-LAMP lumenal domain. These data show that DC-LAMP-mediated antigen targeting is absolutely required for optimal T cell stimulation, but that in our experimental setup, the lumenal part of DC-LAMP does not improve the overall induction of antigen-specific T cell responses.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Antigen Presentation
  • Cells, Cultured
  • Dendritic Cells / immunology*
  • Dendritic Cells / metabolism
  • Electroporation*
  • Gene Products, gag / genetics
  • Gene Products, gag / immunology*
  • Gene Products, gag / metabolism
  • HIV Infections / immunology
  • HIV Infections / virology
  • HIV-1 / immunology
  • Humans
  • Lymphocyte Activation
  • Lysosomal Membrane Proteins* / chemistry
  • Lysosomal Membrane Proteins* / genetics
  • Lysosomal Membrane Proteins* / immunology
  • Lysosomal Membrane Proteins* / metabolism
  • RNA, Messenger / genetics
  • RNA, Messenger / metabolism*
  • T-Lymphocytes / immunology*

Substances

  • Gene Products, gag
  • Lysosomal Membrane Proteins
  • RNA, Messenger