Induction of human immunodeficiency virus type 1 expression in chronically infected cells is associated primarily with a shift in RNA splicing patterns

J Virol. 1991 Mar;65(3):1291-303. doi: 10.1128/JVI.65.3.1291-1303.1991.

Abstract

We have analyzed the kinetics of human immunodeficiency virus type 1 (HIV-1) RNA induction in chronically infected T cells and promonocytes. A substantial amount of spliced mRNAs and assembled virions was found in resting cells. Induction increased the steady-state level of total HIV-1 RNA by 4-fold but increased the level of unspliced transcripts by 25-fold. This increase in unspliced RNA was reflected in the amount of virus seen by electron microscopy. These data suggest a mechanism for the induction of HIV-1 RNA in chronically infected cells involving a shift in splicing greatly favoring the stability of unspliced viral RNA with only a modest increase in total viral RNA. Analysis of the relative abundance of transcript classes is critical to the measurement of HIV-1 viral replication kinetics.

MeSH terms

  • Base Sequence
  • Cell Line
  • Cell Transformation, Viral*
  • HIV-1 / genetics*
  • HIV-1 / ultrastructure
  • Humans
  • Kinetics
  • Microscopy, Electron
  • Molecular Sequence Data
  • Oligonucleotide Probes
  • Polymerase Chain Reaction
  • RNA Splicing*
  • RNA, Messenger / genetics*
  • RNA, Viral / biosynthesis
  • RNA, Viral / genetics*
  • RNA, Viral / ultrastructure
  • T-Lymphocytes
  • Transcription, Genetic*
  • Virion / genetics

Substances

  • Oligonucleotide Probes
  • RNA, Messenger
  • RNA, Viral