Interleukin-21 restrains tumor growth and induces a substantial increase in the number of circulating tumor-specific T cells in a murine model of malignant melanoma

Cytokine. 2010 Jan;49(1):80-8. doi: 10.1016/j.cyto.2009.11.001. Epub 2009 Dec 3.

Abstract

New strategies of immunotherapy are currently being evaluated, and the combination of chemo- and immunotherapy has shown promising results. The cytokine interleukin-21 (IL-21) is known to enhance immune function, and in this study we have investigated its ability to boost the efficacy of chemoimmunotherapy-cyclophosphamide and adoptive cell transfer (ACT)-in the B16-OVA/OT-I murine model of malignant melanoma. Subcutaneous B16-OVA tumors were established in C57BL/6J mice 8 days before adoptive transfer of tumor-specific OT-I T cells. In addition to cyclophosphamide and ACT, one group of mice received daily injections of murine IL-21 (mIL-21). Mice treated with mIL-21 had more tumor-specific T cells in the circulation 4 and 7 days following ACT (P=0.004 and P=0.002, respectively). Importantly, mIL-21 and ACT controlled tumor growth instantly and more effectively than ACT alone (P=0.001, day 4)-an effect that persisted up to 5 days after the last mIL-21 injection. We conclude that mIL-21 enhances chemoimmunotherapy: it amplifies the number of tumor-specific T cells in the circulation and also stunts early tumor growth.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Adoptive Transfer
  • Animals
  • Antineoplastic Agents, Alkylating / pharmacology
  • Antineoplastic Agents, Alkylating / therapeutic use
  • Cyclophosphamide / pharmacology
  • Cyclophosphamide / therapeutic use
  • Disease Models, Animal
  • Female
  • Immunotherapy / methods
  • Interleukin-21
  • Interleukins* / immunology
  • Interleukins* / therapeutic use
  • Kaplan-Meier Estimate
  • Lysosomal Membrane Proteins / metabolism
  • Melanoma* / drug therapy
  • Melanoma* / immunology
  • Melanoma* / pathology
  • Melanoma, Experimental* / drug therapy
  • Melanoma, Experimental* / immunology
  • Melanoma, Experimental* / pathology
  • Mice
  • Mice, Inbred C57BL
  • Mice, Transgenic
  • Spleen / cytology
  • Spleen / immunology
  • T-Lymphocytes / drug effects
  • T-Lymphocytes / immunology*
  • T-Lymphocytes / transplantation

Substances

  • Antineoplastic Agents, Alkylating
  • Interleukins
  • Lamp1 protein, mouse
  • Lysosomal Membrane Proteins
  • Cyclophosphamide
  • Interleukin-21