The role of SRC-3 in human breast cancer

Nat Rev Clin Oncol. 2010 Feb;7(2):83-9. doi: 10.1038/nrclinonc.2009.219. Epub 2009 Dec 22.

Abstract

Members of the nuclear receptor superfamily are ligand-regulated transcription factors involved in the control of a broad range of normal physiological and disease processes. The estrogen receptor alpha (ERalpha) is a member of the steriod receptor family, which is part of the nuclear receptor superfamily. ERalpha it is important for many biological processes and plays a key role in the pathogenesis of breast cancer. Gene regulation by ERalpha requires the recruitment of a multitude of transcriptional co-regulators to the promoters of estrogen-responsive genes. There is evidence in support of the involvement of these co-regulators in breast cancer progression. We review the role of steroid receptor co-activator-3 (SRC-3), which is frequently amplified in breast cancer, and its role in breast cancer risk, outcome and response to endocrine therapy in patients with breast cancer.

Publication types

  • Research Support, Non-U.S. Gov't
  • Review

MeSH terms

  • Breast Neoplasms / genetics*
  • Estrogen Receptor alpha / metabolism*
  • Female
  • Gene Expression Regulation, Neoplastic*
  • Humans
  • Nuclear Receptor Coactivator 3 / metabolism*

Substances

  • Estrogen Receptor alpha
  • NCOA3 protein, human
  • Nuclear Receptor Coactivator 3