[The inhibitory effect of curcumin on the proliferation of HT-29 colonic cancer cell induced by deoxycholic acid]

Zhonghua Nei Ke Za Zhi. 2009 Sep;48(9):760-3.
[Article in Chinese]

Abstract

Objective: To assess the effect of curcumin on deoxycholic acid (DCA) induced cell proliferation in human HT-29 colon cancer cell line and to discuss its underlying anti-cancer mechanisms.

Methods: The effect of curcumin on cell proliferation was tested in vitro with MTT assay. RT-PCR was applied to measure cyclooxygenase-2 (COX-2) mRNA transcription. Cellular immunochemical stain was applied to label COX-2 protein expression. Radio immunoassay was applied to assess prostaglandin E(2) (PGE(2)) level.

Results: DCA increased cell proliferation, COX-2 mRNA expression and PGE(2) concentration of HT-29 cell line. Curcumin inhibited DCA-induced cell proliferation of HT-29 cell in a dose and time depended manner. Curcumin may inhibit DCA-induced COX-2 mRNA transcription at 6 hour with more than 5 micromol/L and at 12 hour with more than 0.5 micromol/L. However, Curcumin may decrease DCA-induced COX-2 expression and PGE(2) synthesis at 24 - 48 hour with more than 1 micromol/L and at 6-12 hour with more than 5 micromol/L.

Conclusions: Curcumin may inhibit HT-29 cell proliferation, COX-2 mRNA transcription, COX-2 protein expression and PGE(2) synthesis induced by DCA in HT-29 cell line. These data provide new insights into the mechanism of its anti-cancer properties.

Publication types

  • English Abstract

MeSH terms

  • Apoptosis
  • Cell Proliferation / drug effects*
  • Curcumin / pharmacology*
  • Cyclooxygenase 2 / metabolism*
  • Deoxycholic Acid
  • Dinoprostone / biosynthesis
  • HT29 Cells
  • Humans

Substances

  • Deoxycholic Acid
  • Cyclooxygenase 2
  • PTGS2 protein, human
  • Curcumin
  • Dinoprostone