Post-transcriptional regulation of cytoskeletal actin and T lymphocyte receptor beta chain mRNA by phorbol ester

Biochim Biophys Acta. 1991 Mar 19;1092(1):124-7. doi: 10.1016/0167-4889(91)90186-2.

Abstract

The protein kinase C activator phorbol 12-myristate 13-acetate (TPA) augments the level of cytoplasmic mRNA for cytoskeletal actin and the beta chain of the T cell antigen receptor (TCR beta) in peripheral blood lymphocytes. Calcium ionophore A23187 has an antagonistic effect on these increases. The immunosuppressive drug cyclosporin A blocked both positive and negative effects of A23187 without affecting those of TPA. TPA-mediated increases in cytoplasmic RNA for both genes can occur when transcription is blocked with actinomycin D and no increase is observed in the corresponding total cellular RNA. Activation of protein kinase C can thus mediate the accumulation of these mRNAs by a post-transcriptional mechanism.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Actins / genetics*
  • Calcimycin / pharmacology
  • Cyclosporins / pharmacology
  • Cytoplasm / drug effects
  • Cytoplasm / metabolism
  • Dactinomycin / pharmacology
  • Gene Expression Regulation / drug effects*
  • Humans
  • Protein Kinase C / metabolism
  • RNA, Messenger / metabolism*
  • Receptors, Antigen, T-Cell / genetics*
  • Tetradecanoylphorbol Acetate / pharmacology*
  • Transcription, Genetic

Substances

  • Actins
  • Cyclosporins
  • RNA, Messenger
  • Receptors, Antigen, T-Cell
  • Dactinomycin
  • Calcimycin
  • Protein Kinase C
  • Tetradecanoylphorbol Acetate