[The effect of simultaneously blocking target epidermal growth factor receptor tyrosine kinase and cyclooxygenase-2 on the growth of NPC cell]

Lin Chuang Er Bi Yan Hou Tou Jing Wai Ke Za Zhi. 2009 Sep;23(18):817-20, 823.
[Article in Chinese]

Abstract

Objective: This study was designed to prove simultaneously blocking both EGFR and COX-2-mediated pathways may be an efficient means of inhibiting cancer cell growth in NPC.

Method: A combination of tarceva (EGFR-selective tyrosine kinase inhibitors) with celecoxib (Cox-2 inhibitor) was studied on its effects on cell growth, cell cycle progression, apoptosis and protein expression in CNE-2 cell lines by cell growth assay, flow cytometric analysis assay and Western blotting.

Result: (1) The inhibition rate of cell growth was higher in the group treated with a combination of two agents than that the sum of rates of the two groups treated with only one agent (P < 0.05). (2) The combination of tarceva with celecoxib significantly induced G1 arrest (P < 0.05), but did not increase apoptosis rate (P > 0.05). (3) The group of combination showed less expressions of p-EGFR and COX-2 than any other group.

Conclusion: Simultaneously blocking EGFR and COX-2 mediated pathways would significantly inhibit the growth of CNE-2 cell line, increase G1 arrest and reduce the expression levels of p-EGFR and COX-2.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Apoptosis
  • Celecoxib
  • Cell Division
  • Cyclooxygenase 2 / metabolism*
  • ErbB Receptors / metabolism*
  • Erlotinib Hydrochloride
  • Humans
  • Pyrazoles / administration & dosage
  • Pyrazoles / pharmacology*
  • Quinazolines / administration & dosage
  • Quinazolines / pharmacology*
  • Signal Transduction
  • Sulfonamides / administration & dosage
  • Sulfonamides / pharmacology*
  • Tumor Cells, Cultured

Substances

  • Pyrazoles
  • Quinazolines
  • Sulfonamides
  • Erlotinib Hydrochloride
  • Cyclooxygenase 2
  • PTGS2 protein, human
  • EGFR protein, human
  • ErbB Receptors
  • Celecoxib