[Ursodeoxycholic acid inhibits hepatocyte-like cell apoptosis by down-regulating the expressions of Bax and Caspase-3]

Zhonghua Yi Xue Za Zhi. 2009 Nov 17;89(42):2997-3001.
[Article in Chinese]

Abstract

Objective: To investigate the mechanism of ursodeoxycholic acid (UDCA) in hepatocyte apoptosis using differentiated hepatocytes derived from bone marrow mesenchymal cells.

Methods: Rat bone marrow mesenchymal cell was induced into mature hepatocytes in vitro and then treated with PBS (Cont), deoxycholic acid (DCA), DCA plus UDCA (U + D) or UDCA alone (UDCA). Cell apoptosis was detected by Hoechst staining and Caspase-3 activity measurement. The mRNA expressions of p53 and Bax were measured by semi-quantitative RT-PCR and real-time RT-PCR. The Bax protein expression was detected by immunohistochemistry.

Results: In comparison with Cont, DCA obviously induced hepatocyte apoptosis as measured by an increased cell count and a higher Caspase-3 activity (P < 0.05). These increments could be inhibited by addition of UDCA. The expressions of p53 and Bax in hepatocytes were up-regulated by DCA. These up-expressions could also be inhibited by UDCA. The DCA-induced increased count of Bax-positive cells could be reduced by UDCA.

Conclusion: UDCA inhibits DCA-induced hepatocyte apoptosis by down-regulating the expression of p53/Bax signal molecule.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Animals
  • Apoptosis / drug effects*
  • Bone Marrow Cells
  • Caspase 3 / metabolism*
  • Cell Differentiation
  • Cells, Cultured
  • Down-Regulation
  • Female
  • Hepatocytes / cytology
  • Hepatocytes / drug effects*
  • Hepatocytes / metabolism
  • Mesenchymal Stem Cells / cytology
  • Rats
  • Rats, Sprague-Dawley
  • Ursodeoxycholic Acid / pharmacology*
  • bcl-2-Associated X Protein / metabolism*

Substances

  • Bax protein, rat
  • bcl-2-Associated X Protein
  • Ursodeoxycholic Acid
  • Casp3 protein, rat
  • Caspase 3