Abstract
Kostmann disease is a rare autosomal recessive form of severe congenital neutropenia characterized by maturation arrest at the stage of promyelocytes/myelocytes in bone marrow with peripheral blood absolute neutrophil counts below 0.5 x 10(9)/L and severe recurrent bacterial infections from early infancy. Kostmann disease is caused by homozygous mutations in the gene encoding the mitochondrial protein HCLS1-associated X1. Here, we report three patients with Kostmann disease who, besides recurrent infections, have developmental delay.
MeSH terms
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Adaptor Proteins, Signal Transducing / deficiency*
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Adaptor Proteins, Signal Transducing / genetics
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Age of Onset
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Child, Preschool
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Consanguinity
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Developmental Disabilities* / diagnosis
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Developmental Disabilities* / genetics
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Developmental Disabilities* / therapy
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Female
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Granulocyte Colony-Stimulating Factor / therapeutic use
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Humans
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Immunologic Deficiency Syndromes* / diagnosis
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Immunologic Deficiency Syndromes* / genetics
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Immunologic Deficiency Syndromes* / therapy
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Infant
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Male
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Neutropenia / congenital*
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Neutropenia / diagnosis
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Neutropenia / genetics
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Neutropenia / therapy
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Syndrome
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Turkey
Substances
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Adaptor Proteins, Signal Transducing
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HAX1 protein, human
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Granulocyte Colony-Stimulating Factor