Acute food deprivation reduces expression of diazepam-binding inhibitor, the precursor of the anorexigenic octadecaneuropeptide ODN, in mouse glial cells

J Mol Endocrinol. 2010 May;44(5):295-9. doi: 10.1677/JME-09-0176. Epub 2010 Mar 10.

Abstract

In the central nervous system of mammals, the gene encoding diazepam-binding inhibitor (DBI) is exclusively expressed in glial cells. Previous studies have shown that central administration of a DBI processing product, the octadecaneuropeptide ODN, causes a marked inhibition of food consumption in rodents. Paradoxically, however, the effect of food restriction on DBI gene expression has never been investigated. Here, we show that in mice, acute fasting dramatically reduces DBI mRNA levels in the hypothalamus and the ependyma bordering the third and lateral ventricles. I.p. injection of insulin, but not of leptin, selectively stimulated DBI expression in the lateral ventricle area. These data support the notion that glial cells, through the production of endozepines, may relay peripheral signals to neurons involved in the central regulation of energy homeostasis.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Animals
  • Diazepam Binding Inhibitor / metabolism*
  • Down-Regulation
  • Ependyma / metabolism
  • Fasting*
  • Hypothalamus / metabolism
  • Injections, Intraperitoneal
  • Insulin / administration & dosage
  • Lateral Ventricles / metabolism
  • Leptin / administration & dosage
  • Male
  • Mice
  • Neuroglia / metabolism*
  • Neuropeptides / genetics
  • Neuropeptides / metabolism*
  • Peptide Fragments / genetics
  • Peptide Fragments / metabolism*
  • Protein Binding
  • Third Ventricle / metabolism
  • Transcription, Genetic

Substances

  • Diazepam Binding Inhibitor
  • Insulin
  • Leptin
  • Neuropeptides
  • Peptide Fragments
  • diazepam binding inhibitor (33-50)