Tyrosine phosphorylation is essential for microfilament assembly in B lymphocytes

Biochem Biophys Res Commun. 1991 May 15;176(3):1424-9. doi: 10.1016/0006-291x(91)90445-d.

Abstract

The B cell antigen receptor regulates the tyrosine kinase signal transduction pathway and it mediates a variety of morphological changes such as capping and membrane ruffling. The relationship between these two events is unclear. We show here that cross-linking the antigen receptor on human B lymphocytes, in addition to increasing tyrosine phosphorylation of specific substrates, induces the conversion of G-actin to F-actin. Preincubation of B lymphocytes with two different tyrphostins blocked anti-IgM-induced tyrosine phosphorylation and actin polymerization. The ability of the tyrphostins to block anti-IgM induced conversion of G-actin to F-actin indicates that a tyrosine kinase acts as an essential link between the B cell antigen receptor the early changes in cytoskeletal reorganization.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Actin Cytoskeleton / physiology*
  • Actin Cytoskeleton / ultrastructure
  • Actins / analysis
  • Actins / metabolism
  • Antibodies, Anti-Idiotypic / immunology
  • B-Lymphocytes / immunology
  • B-Lymphocytes / physiology*
  • B-Lymphocytes / ultrastructure
  • Humans
  • Immunoglobulin M / immunology
  • Nitriles / pharmacology
  • Nitrophenols / pharmacology
  • Phenols / pharmacology
  • Phosphorylation
  • Protein-Tyrosine Kinases / metabolism*
  • Receptors, Antigen, B-Cell / physiology
  • Tyrosine
  • Tyrphostins*

Substances

  • Actins
  • Antibodies, Anti-Idiotypic
  • Immunoglobulin M
  • Nitriles
  • Nitrophenols
  • Phenols
  • Receptors, Antigen, B-Cell
  • Tyrphostins
  • AG 127
  • AG 30
  • AG 183
  • Tyrosine
  • Protein-Tyrosine Kinases