Regulatory mechanisms for apoptosis in placental tissue during normal pregnancy and gestosis-complicated pregnancy

Bull Exp Biol Med. 2009 Nov;148(5):766-70. doi: 10.1007/s10517-010-0812-8.

Abstract

The localization of apoptosis and expression of proapoptotic and antiapoptotic factors by the placental tissue were compared during normal pregnancy and gestosis-complicated pregnancy. The degree of apoptosis did not differ in the third trimester of normal pregnancy and gestosis-complicated pregnancy. Increased expression of Fas, caspase-8, and caspase-3 in placental tissue during normal pregnancy was shown to contribute to the suppression of angiogenesis and growth of placental tissue. No differences were found in the expression of FasL (CD95L), caspase-2, caspase-9, and Mcl-1 by placental cells during normal pregnancy and gestosis-complicated pregnancy. Increased expression of TRAIL by trophoblast cells is a protective mechanism from apoptotic signals of maternal cytotoxic lymphocytes and NK cells during gestosis.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Apoptosis / physiology*
  • Biomarkers / metabolism
  • Caspase 2 / metabolism
  • Caspase 9 / metabolism
  • Fas Ligand Protein / metabolism
  • Female
  • Gestational Age
  • Humans
  • Myeloid Cell Leukemia Sequence 1 Protein
  • Placenta* / pathology
  • Placenta* / physiology
  • Pre-Eclampsia* / pathology
  • Pre-Eclampsia* / physiopathology
  • Pregnancy
  • Proto-Oncogene Proteins c-bcl-2 / metabolism

Substances

  • Biomarkers
  • Fas Ligand Protein
  • Myeloid Cell Leukemia Sequence 1 Protein
  • Proto-Oncogene Proteins c-bcl-2
  • Caspase 2
  • Caspase 9