Histone deacetylase inhibition as a mechanism for the therapeutic effect of statins (3-hydroxy-3-methylglutaryl-coenzyme a reductase inhibitors) in follicular lymphoma

J Clin Oncol. 2010 Aug 20;28(24):e426; author reply e427-8. doi: 10.1200/JCO.2010.28.9488. Epub 2010 Jun 21.
No abstract available

Publication types

  • Comment

MeSH terms

  • Antibodies, Monoclonal / pharmacology*
  • Antibodies, Monoclonal / therapeutic use
  • Antibodies, Monoclonal, Murine-Derived
  • Antineoplastic Agents / antagonists & inhibitors
  • Antineoplastic Agents / pharmacology*
  • Antineoplastic Agents / therapeutic use
  • Clinical Trials as Topic
  • Depsipeptides / pharmacology
  • Disease-Free Survival
  • Drug Interactions
  • Histone Deacetylase Inhibitors / pharmacology*
  • Humans
  • Hydroxamic Acids / pharmacology
  • Hydroxymethylglutaryl-CoA Reductase Inhibitors / pharmacology*
  • Lymphoma, Follicular / drug therapy*
  • Lymphoma, Follicular / metabolism
  • Lymphoma, Non-Hodgkin / drug therapy
  • Rituximab
  • Vorinostat

Substances

  • Antibodies, Monoclonal
  • Antibodies, Monoclonal, Murine-Derived
  • Antineoplastic Agents
  • Depsipeptides
  • Histone Deacetylase Inhibitors
  • Hydroxamic Acids
  • Hydroxymethylglutaryl-CoA Reductase Inhibitors
  • Rituximab
  • Vorinostat
  • romidepsin