Hypoxia-induced short-term potentiation of respiratory-modulated facial motor output in the rat

Respir Physiol Neurobiol. 2010 Aug 31;173(1):107-11. doi: 10.1016/j.resp.2010.06.015. Epub 2010 Jul 1.

Abstract

Respiratory-modulated facial (VII) nerve discharge includes pre-inspiratory (Pre-I) and inspiratory (I) components. Tonic VII bursting is also present across the respiratory cycle. We tested the hypothesis that hypoxia-induced plasticity of VII motor activity is differentially expressed in Pre-I, I and tonic bursting. Phrenic and VII neurograms were recorded in urethane-anesthetized, vagotomized and ventilated adult rats. A 3 min isocapnic hypoxic challenge (PaO(2)=33+/-2 mmHg) was used to evoke respiratory short-term potentiation (STP). Pre-I, I and tonic VII activity increased immediately at the initial stage of hypoxia (i.e. acute response) and then progressively increased as hypoxia was maintained. Following hypoxia, I VII activity remained elevated (i.e. post-hypoxia STP) but both Pre-I and tonic activity immediately returned to baseline values. We conclude that STP following hypoxia is preferentially expressed in I compared to Pre-I and tonic VII activity.

Publication types

  • Research Support, N.I.H., Extramural
  • Research Support, Non-U.S. Gov't
  • Research Support, U.S. Gov't, Non-P.H.S.

MeSH terms

  • Action Potentials / physiology
  • Animals
  • Blood Gas Analysis / methods
  • Blood Pressure / physiology
  • Hypoxia / physiopathology*
  • Motor Neurons / physiology*
  • Phrenic Nerve / physiopathology*
  • Rats
  • Rats, Sprague-Dawley
  • Respiration*
  • Time Factors
  • Vagotomy / methods