TAT-mediated transduction of NF-Ya peptide induces the ex vivo proliferation and engraftment potential of human hematopoietic progenitor cells

Blood. 2010 Oct 14;116(15):2676-83. doi: 10.1182/blood-2010-03-273441. Epub 2010 Jul 8.

Abstract

Retroviral overexpression of NF-Ya, the regulatory subunit of the transcription factor NF-Y, activates the transcription of multiple genes implicated in hematopoietic stem cell (HSC) self-renewal and differentiation and directs HSCs toward self-renewal. We asked whether TAT-NF-Ya fusion protein could be used to transduce human CD34(+) cells as a safer, more regulated alternative approach to gene therapy. Here we show that externally added recombinant protein was able to enter the cell nucleus and activate HOXB4, a target gene of NF-Ya, using real-time polymerase chain reaction RNA and luciferase-based protein assays. After TAT-NF-Ya transduction, the proliferation of human CD34(+) cells in the presence of myeloid cytokines was increased 4-fold. Moreover, TAT-NF-Ya-treated human primary bone marrow cells showed a 4-fold increase in the percentage of huCD45(+) cells recovered from the bone marrow of sublethally irradiated, transplanted NOD-Scid IL2Rγ(null) mice. These data demonstrate that TAT-peptide therapies are an alternative approach to retroviral stem cell therapies and suggest that NF-Ya peptide delivery should be further evaluated as a tool for HSC/progenitors ex vivo expansion and therapy.

Publication types

  • Research Support, N.I.H., Extramural

MeSH terms

  • Animals
  • CCAAT-Binding Factor / genetics*
  • CCAAT-Binding Factor / metabolism*
  • Cell Proliferation
  • Genetic Therapy
  • Hematopoietic Stem Cell Transplantation
  • Hematopoietic Stem Cells / cytology*
  • Hematopoietic Stem Cells / metabolism*
  • Homeodomain Proteins / genetics
  • Humans
  • In Vitro Techniques
  • Interleukin Receptor Common gamma Subunit / deficiency
  • Interleukin Receptor Common gamma Subunit / genetics
  • Mice
  • Mice, Inbred NOD
  • Mice, Knockout
  • Mice, SCID
  • Peptide Fragments / genetics*
  • Peptide Fragments / metabolism*
  • Promoter Regions, Genetic
  • Protein Binding
  • Recombinant Fusion Proteins / genetics
  • Recombinant Fusion Proteins / metabolism
  • Transcription Factors / genetics
  • Transcriptional Activation
  • Transduction, Genetic*
  • Transplantation, Heterologous
  • tat Gene Products, Human Immunodeficiency Virus / genetics*
  • tat Gene Products, Human Immunodeficiency Virus / metabolism*

Substances

  • CCAAT-Binding Factor
  • HOXB4 protein, human
  • Homeodomain Proteins
  • Interleukin Receptor Common gamma Subunit
  • NFYA protein, human
  • Peptide Fragments
  • Recombinant Fusion Proteins
  • Transcription Factors
  • tat Gene Products, Human Immunodeficiency Virus
  • tat peptide (47-57), Human immunodeficiency virus 1