Protective effects of kappa-ca3000+CP against ultraviolet-induced damage in HaCaT and MEF cells

J Photochem Photobiol B. 2010 Oct 5;101(1):22-30. doi: 10.1016/j.jphotobiol.2010.06.007. Epub 2010 Jun 30.

Abstract

In this study, the complex kappa-ca3000+CP combined collagen peptide with kappa-carrageenan oligosaccharide was tested for its ability to moderate UV-induced damage and investigated for its protective mechanism against UV radiation. Human keratinocytes (HaCaT) and mouse embryonic fibroblasts (MEF) were used to monitor the effects of kappa-ca3000+CP on cell viability, apoptosis, level of collagen I and MMP-1, MAPKs activation and intracellular ROS production after UV-irradiation. The results indicated that application of the kappa-ca3000+CP (100 microg/ml) could significantly attenuate UV-induced HaCaT and MEF death, as well as inhibit the UV-induced apoptosis of HaCaT cells. The decreased collagen I synthesis and the increased MMP-1 expression of MEF by UV radiation were almost restored back to normal level after treatment with kappa-ca3000+CP. Moreover, kappa-ca3000+CP could significantly suppress UV-induced MAPKs activation and intracellular ROS production. Taken together, these results showed that antioxidant property of kappa-ca3000+CP can effectively attenuate UV-caused cell damage and skin photoaging by suppressing cell apoptosis and expression of MMP-1 through the MAPKs signaling pathways. Thus, kappa-ca3000+CP has potential antiaging effects and prominent protective effects on UV-induced skin cell damages, which might be used in pharmaceutical and cosmetic industries.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Animals
  • Apoptosis / radiation effects
  • Carrageenan / chemistry
  • Carrageenan / pharmacology*
  • Cells, Cultured
  • Cellular Senescence
  • Collagen / pharmacology*
  • Collagen Type I / metabolism
  • Fibroblasts / drug effects*
  • Humans
  • Keratinocytes / drug effects*
  • Matrix Metalloproteinase 1 / metabolism
  • Mice
  • Mitogen-Activated Protein Kinase Kinases / metabolism
  • Reactive Oxygen Species / metabolism
  • Ultraviolet Rays*

Substances

  • Collagen Type I
  • Reactive Oxygen Species
  • Carrageenan
  • Collagen
  • Mitogen-Activated Protein Kinase Kinases
  • Matrix Metalloproteinase 1